i by myself have consented to use ter neoterm Dysendotheliosis.
is collapsing gp caused by proximal afferent swelling with secondary hypoperfusion and low pressure squeezout
if so all the podo diseases with effacement should be epireactions to hypoperfused glomeruli ?
lack of nitric oxide synthetase ? hypercontractile podo falling of their adhesions ? disrupting slit membranes ?
time to crave for a pizza to get some glucose into brain. doing stupid tasks copy paste copypaste for site maintenance.