="" Omics of Covid-19 disease – coViki

Omics of Covid-19 disease

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Maybe, specific information might be found in the raw readout of proteomic datasets.

Searchterm applied (gives 9900 papers on june 7th 2022)

http://www.moremed.org/SiteSearch.php?S1=transcriptom*+OR+transcription*+OR+proteom*+OR+gene+expression+OR+interactom*+OR+omics+OR+hub+genes+OR+epigenom*+OR+dna+methylation+OR+metabolom*+OR+gene+chip+OR+transcripts+OR+microarray+OR+receptorom*+OR+glycom*+OR+differential+expression&S2=covid19+OR+covid-19+OR+sars-cov2+OR+sars-cov-2+&S3=&submit=submit&px1=proxy.nationallizenzen.de&px2=&px3=&px4=&R=all&Y0=1500&Y1=2025

Covid Omics Databases and Webservers


34845454 2021. Identifying disease-critical cell types and cellular processes across the human body by integration of single-cell profiles and human genetics.
34127975 2021. Implicating Gene and Cell Networks Responsible for Differential COVID-19 Host Responses via an Interactive Single Cell Web Portal.
34075382 2021. CovidExpress: an interactive portal for intuitive investigation on SARS-CoV-2 related transcriptomes.
32587962 2020. The IMEx Coronavirus interactome: an evolving map of Coronaviridae-Host molecular interactions.
33413085 2021. H2V: a database of human genes and proteins that respond to SARS-CoV-2, SARS-CoV, and MERS-CoV infection.
34448623 2021. A Novel Pathway Network Analytics Method Based on Graph Theory.
33605735 2021. Open Science Resources for the Mass Spectrometry-Based Analysis of SARS-CoV-2.
32930583 2020. Quantification of mRNA Expression Using Single-Molecule Nanopore Sensing.
33553941 2021. SARSCOVIDB-A New Platform for the Analysis of the Molecular Impact of SARS-CoV-2 Viral Infection.


33501381 2020. Fostering global data sharing: highlighting the recommendations of the Research Data Alliance COVID-19 working group.

-to be shown-

All relevant papers


34920080 2022. The spike protein of SARS-CoV-2 induces heme oxygenase-1: Pathophysiologic implications.
34835015 2021. SARS-CoV-2 Spike Protein S1-Mediated Endothelial Injury and Pro-Inflammatory State Is Amplified by Dihydrotestosterone and Prevented by Mineralocorticoid Antagonism. -to be shown-

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